Dr. Paul Mason on the link between seed oils and sunburn
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Dr Paul Mason obtained his medical degree with honours from the University of Sydney, and also holds degrees in Physiotherapy and Occupational Health. He is ...

Do seed oils block cholesterol to vitamin D? Vitamin D as sunscreen Sunburn resistance of people who don’t eat seed oils

Summary by Google's LLM

In this video clip from Low Carb Down Under, Dr. Paul Mason presents a theory linking the consumption of industrial seed oils to a higher susceptibility to sunburn

Key Arguments and Claims:

  • Vitamin D as Natural Sunscreen: Dr. Mason states that the body naturally vitamin D as a protective shield against UV/UVB radiation damage to DNA, rather than strictly for bone health
  • The Cholesterol Connection: He references Ancel Keys’ historical “Seven Countries Study”, highlighting a data point that individuals with higher sun exposure had lower blood cholesterol levels. He explains this occurs because the body uses cholesterol to synthesize vitamin D
  • Interference by Plant Sterols: He argues that plant sterols (phytosterols) absorbed from dietary seed oils interfere with normal cholesterol chemistry, specifically disrupting the skin’s ability to synthesize vitamin D
  • Anecdotal Evidence: While acknowledging the evidence is largely anecdotal, he points out that a vast number of individuals on ketogenic internet forums report a noticeable resistance to sunburn after completely eliminating seed oils from their diet.
  • Pommes_für_dein_Balg@feddit.orgEnglish
    3·
    8 days ago

    The study you linked is about the effect of phytosterols on Vitamin D adsorption from an oral source.
    It says nothing about Vitamin D production from sunlight.
    And the study involved a single dose of Vitamin D given, to 40 people.

    Long term (> 4 weeks) administration of sterols is needed to investigate the impact on Vitamin D absorption and consequently its metabolism.

    So the study itself says “for actual evidence, we’d need more data”.

    • jet@hackertalks.comMEnglish
      4·
      7 days ago

      The study you linked is about the effect of phytosterols on Vitamin D adsorption from an oral source. It says nothing about Vitamin D production from sunlight.

      The point was to demonstrate plant sterols have a impact on the bodies cholesteral systems including vitamin d production and absorption.

      So the study itself says “for actual evidence, we’d need more data”.

      Literally every paper ever written says that. We always need more studies, that’s the nature of curiosity. In fact professors have to admonish grad students not to add that to papers since its just filler at this point.

      I’m not sure what your core thesis is: Vitamin D is not relevant in sun exposure?

      https://doi.org/10.1016/j.jid.2017.04.040 -

      Oral Vitamin D Rapidly Attenuates Inflammation from Sunburn: An Interventional Study

      The diverse immunomodulatory effects of vitamin D are increasingly being recognized. However, the ability of oral vitamin D to modulate acute inflammation in vivo has not been established in humans. In a double-blinded, placebo-controlled interventional trial, 20 healthy adults were randomized to receive either placebo or a high dose of vitamin D3 (cholecalciferol) one hour after experimental sunburn induced by an erythemogenic dose of UVR. Compared with placebo, participants receiving vitamin D3 (200,000 international units) demonstrated reduced expression of proinflammatory mediators tumor necrosis factor-α (P = 0.04) and inducible nitric oxide synthase (P = 0.02) in skin biopsy specimens 48 hours after experimental sunburn. A blinded, unsupervised hierarchical clustering of participants based on global gene expression profiles revealed that participants with significantly higher serum vitamin D3 levels after treatment (P = 0.007) demonstrated increased skin expression of the anti-inflammatory mediator arginase-1 (P = 0.005), and a sustained reduction in skin redness (P = 0.02), correlating with significant expression of genes related to skin barrier repair. In contrast, participants with lower serum vitamin D3 levels had significant expression of proinflammatory genes. Together the data may have broad implications for the immunotherapeutic properties of vitamin D in skin homeostasis, and implicate arginase-1 upregulation as a previously unreported mechanism by which vitamin D exerts anti-inflammatory effects in humans.

      Yes, its oral, but it demonstrates that vitamin D is a necessary part of the bodies sun exposure mechanisms. i.e. its produced on demand to treat the damage the sun is doing to the skin.